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  • Studi Relativi al Potere Antiacido del Bicarbonato di Sodio nei Tumori (Dr. Tullio Simoncini, oncologo, http://www.curenaturalicancro.org)

    Ricerca personalizzata

    Mi manda Rai 3 ha ancora parlato di Simoncini che ora opera in Olanda diffondendo il suo metodo basato sul bicarbonato di sodio.
    Gli esperti hanno confermato che Simoncini è stato condannato e radiato dall' ordine dei medici. Egli appliccherebbe un metodo rischioso e senza base scientifica. Questo trattamento sarebbe talvolta invasivo e comunque dannoso. In genere poi il paziente abbandonerebbe altri metodi di cura e la malattia generalmente procederebbe più rapidamente, si soffrirebbe e si morirebbe prima


  • 26/05/2006 Aggiornamento sulla Teoria del Cancro e Fungo
    Il bicarbonato di sodio nella cura del cancro non avrebbe alcun effetto...


  • Anne McLean, “Malignant gliomas display altered pH regu1ation byè NHE1 compared with non transformed astrocytes  (Am J Physiol Cell Physiol 278: C676-C688, 2000).

    Marion Stubbs, “Causes and consequences of tumour acidity and implications for treatment”, Molecular Medicine: Today, January 2000 (vol.6).

    Robert J. Gillies, “Causes and consequences of hypoxia and acidity in turners - Novartis Foundatíon symposium”, Molecular Medicine Vol.7 N° 2 February 2001; “Causes and consequences of hypoxia and acìdity in tumour microenvironments”.

    J.R. Griffiths, “Causes and consequences of hypoxia and acìdity in tumour microenvironments”, Glia 1994 Nov:12(3):196-210.

    Tannock, I.F., “Acid pH in tumors and its potential for therapeutic exploitation”, Cancer Res 1989 Aug 15;49(16):4373-84.

    Raghunand, N., “Enhancement of chemotherapy by manipulation of tumour pH”, Br J Cancer 1999 Jun;80(7):1005-11.

    Davydova, I.G., “Dynamics of bioelectric activity of the brain and erythrocyte ultrastructure after intravenous infusion of sodium bicarbonate to oncologic patients.” Biull Eksp Biol Med 1992 Apr;113(4):352-5.

    Davydova, I.G., “Characteristics of the effects of artificial alkalosis on electrical activity of the brain and ultrastructure of blood cells in oncologic patients”, Vestn Ross Akad Med Nauk 1995;(4):24-5.

    Star, R.A., “Regulatory volume decrease in the presence of HCO3- by single osteosarcoma cells UMR-106-01”, J Biol Chem 1992 Sep 5;267(25):17665-9.

    LeBoeuf, R.A., “Intracellular acidification is associated with enhanced morphological transformation in Syrian hamster embryo cells”, Cancer Res 1992 Jan 1;52(1):144-8.

    Raghunand, N., “Acute metabolic alkalosis enhances response of C3H mouse mammary tumors to the weak base mitoxantrone.” Neoplasia. 2001 May-Jun;3(3):227-35.

    Raghunand, N., “pH and chemotherapy pH and chemotherapy” Novartis Found Symp. 200 1;240:199-21 l; discussion 265 -8.

    Raghunand, N., “Enhancement of chemotherapy by manipulation of tumour pH.” Br J Cancer. 1999 Jun;80(7):1005-1 I.

    Raghunand, N., “Tumor acidity, ion trapping and chemotherapeutics. IL pll-dependent partition coefficients predict importance of ion trapping on pharmaeokinetics of weakly basic chemotherapeutie agents.” Bíochem Pharmacol. 2003 Oct 1;66(7):1219-29.”

    Mahoney, B.P., “Tumor acidity, ion trapping and chemotherapeutics. I. Acid plì affects the distribution of ehemotherapeutic agents in vitro.” Biochem Phannacol. 2003 Oct 1;66(7):1207-18.

    Schornack, P.A., “Contributions of cell metabolism and H+ diffusion to the acidic pH of tumors.” Neoplasia. 2003 Mar-Apr;5(2):135-45.

    Giffles, R.J., “MRI of the tumor microenvironment.” J Magn Reson Imaging 2002 Dec; 16(6):75 l.

    Torigoe, T., “Vacuolar H(+)-ATPase: funetional mechanisms and potential as a target for cancer chemotherapy.” Anticancer Drugs. 2002 Mar; 13 (3):23 7-43.

    Griffiths, J.R., “Why are cancers acidic? A carrier-mediated diffusion model for H+ transport in the interstitial fluid.” Novartis Found Symp. 200 1;240:46-62; discussion 62-7, 152-3.

    Webb, S.D., “Modelling tumour acidity and invasion.” Novartis Found Symp. 2001;240:169-8 l; discussion 181-5.

    Gillies, R.J., “The tumour microenvironment: causes and consequences of hypoxia and acidity. Introduction.” Novartis Found Symp. 200 1;240:1-6.

    Gillies, R.J., “Causes and consequences of hypoxia and acidity in tumors” Novartis Foundation symposium. Trends Mol Med. 2001 Feb;7(2):47-9.

    Griffiths, JR. “Causes and consequences of hypoxia and acidity in tumour microenvironments. Bioessays. 2001 Mar;23(3):295-6.

    Gillies, R.J., “Causes and effects of heterogeneous perfusion in tumors.” Neoplasia. 1999 Aug; 1 (3):197-207.

    Stubbs, M., “Causes and consequences of tumour acidity and implications for treatment.” Mol Med Today. 2000 Jan;6(1):15-9

    Stubbs, M., “Causes and consequences of acidic ph in tumors: a magnetic resonance study.” Adv. Enzyme Regul. 1999;39;13-30.

    Webb, S.D., “Mathematical modelling of tumour acidity: regulation of intracellular pH.” J Theor Biol. 1999 Jan 21; 196(2);237-50.

    Yamagata, M., “The contribution of lactic acid to acidification of tumours: studies of variant cells lacking lactate dehydrogenase.” Br J Cancer. 1998 Jun;77(11):1726~3 I.

    Martin, G.R., “Non invasive measurement of interstitial pH profiles in normal and neoplastie tissue using fluorescence ratio imaging microscopy.” Cancer Res. 1994 Nov 1;54(21):5670-4.

    Boyer, M.J., “Regulation of intracellular pH in subpopulations of cefis derived from spheroids and solid tumours.” Br J Cancer. 1993 Nov;68(5):890-7.

    Newell, K., “Studies with gIyeolysis-dericient celIs suggest that production of lactic acid is not the only cause of tumor acidity.”  

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